23andMe announces publication of genome-wide association study of motion sickness 23andMe.

Estimates indicate that up to 70 % of variation in risk for motion sickness is due to genetics. Until now there's been an unhealthy understanding of the genetics of movement sickness, despite it being a common condition fairly, stated 23andMe Scientist Bethann Hromatka, lead writer of the study. With the help of 23andMe customers we've had the opportunity to uncover a few of the underlying genetics of the condition. These findings may help provide clues about the sources of motion sickness and various other related circumstances, and how to treat them, which is quite fascinating.Humans do not have chitin in the top of intestinal cells, where the bacterium takes hold, and researchers have been looking for another substance that may be responsible for playing a job in attachment. In the study, Taylor and co-workers screened cultured intestinal cells and discovered mutant bacteria that had problems binding to the intestinal cells. One mutant stress of V. Cholerae lacks a gene that enables it to bind with a sugars called GlcNAc properly. When they compared it with normal, wild-type V. Cholerae bacteria, the researchers found that the protein encoded by this gene supplied normal bacteria the capability to put on the GlcNAc on cells.